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Examining a Patient with Suspected Stroke

Essentially the NIH Stroke Scale is the most targeted, well validated and universally adopted way to examine in suspected AIS.

It takes a bit of practice to become efficient!

The NIHSS is a 15-item neurologic examination stroke scale used to evaluate the effect of acute cerebral infarction on the levels of consciousness, language, neglect, visual-field loss, extraocular movement, motor strength, ataxia, dysarthria, and sensory loss.

A trained observer rates the patient’s ability to answer questions and perform activities. Ratings for each item are scored on a 3- to 5-point scale, with 0 as normal, and there is an allowance for untestable items. Scores range from 0 to 42, with higher scores indicating greater severity.

A single patient assessment requires less than 10 minutes to complete.

The evaluation of stroke severity depends upon the ability of the observer to accurately and consistently assess the patient.

Stroke severity may be stratified on the basis of NIHSS scores as follows:

Very Severe: >25

Severe: 15 – 24

Mild to Moderately Severe: 5 – 14

Mild: 1 – 5

How do you do the NIHSS?

NIHSS Calculator

Here’s an online calculator

VISUAL AIDS

To help you remember it at the bedside!

NIHSS Bedside Booklet
Case Study Icon GandB

Our patient, Jose, has the following findings:

Alert and oriented

Left-sided visual inattention

Left-sided complete homonymous hemianopia

Smile asymmetry on left side of face

Left arm no effort against gravity

Left leg no movement

Right arm and leg normal

No ataxia

Mild sensory loss on left side – less sharp, more dull

Dysarthria, slurring but can be understood

> What is his NIHSS?


1A: 0

1B: 0

1c: 0

2: 0

3: Bilateral hemianopia = +3

4: Minor paralysis (smile asymmetry left side of face) = +1

5A: Left arm no effort against gravity = +3

5B: 0

6A: No movement = +4

6B: 0

7: 0

8: Mild sensory loss – less sharp, more dull on left = +1

9: 0

10: Dysarthria, slurring but can be understood = +1

11: Left sided visual inattention = +1

NIHSS = 14

Stroke Syndromes

Reality is never as clean as these descriptions imply, so don’t get too caught up on this.

LEFT

Right hemiplegia of arms and face (motor + premotor cortex)

Right hemisensory loss of arms and face (somatosensory cortex)

Aphasia (Broca’s area, pars opercularis)

Gaze deviation towards left (frontal eye field)

Right homonymous hemianopia (optic radiation)

Superior division

  • motor cortex
  • Superior visual radiation
  • Broca’s area

Inferior division

  • Wernickes area
  • Inferior visual radiation

RIGHT

Left hemiplegia of arms and face

Left hemisensory loss of arms and face

Left sided neglect (parietal region)

Gaze deviation to the right

Left-sided homonymous hemianopia

Contralateral leg weakness

Contralateral sensory loss of legs 

Cognitive changes e.g. abulia

If recurrent artery of Huebner

  • Contralateral hemiparesis (internal capsule)
  • Movement disorder (caudate head)

Anterior Choroidal Artery

Contralateral visual field defects (lateral geniculate nucleus)

Contralateral hemiparesis (internal capsule)

Contralateral hemisensory loss (internal capsule)

Contralateral homonymous hemianopia (occipital lobe)

Impaired short-term memory (medial temporal/ hippocampus)

Alexia without agraphia (left inferior temporal lobe)

Prosopagnosia (right inferior temporal lobe)

Changes in level of arousal (thalamus)

If artery of Percheron affected:

  • Left and right thalami supplied by single artery from PCA
  • Altered mental status

Corticospinal – limb weakness, often bilateral, limb hyperreflexia, extensor plantar response

Corticobulbar- facial weakness, dysarthria, dysphagia, increased gag reflex

Oculomotor – diplopia, gaze palsies, nystagmus, INO

Reticular activating system- reduced consciousness

Lateral pontine syndrome

Vertigo, vomiting, nystagmus (vestibular nuclei)

Ipsilateral loss of sensation in face (principal sensory trigeminal nucleus)

Facial paralysis (facial nucleus)

Hearing loss, tinnitus (cochlear nucleus)

Lateral medullary syndrome

Contralateral loss of pain and sensation in body (spinothalamic tract)

Ipsilateral facial loss of pain and temperature (spinal trigeminal nucleus)

Dysphasia, dysphonia (nucleus ambiguous)

Vertigo, diplopia, nystagmus, vomiting (inferior vestibular nucleus)

Ipsilateral Horners (sympathetic fibers)

Palatal clonus (central trigeminal tract)

Ataxia (inferior cerebellar peduncle)

Occlusion of small penetrating arteries affecting subcortical matter
Pure motor

  • Posterior limb of internal capsule or anterior pons

Pure sensory

  • VPL / VPM nuclei of thalamus

Ataxia + hemiparesis

  • corticospinal tract + corticopontocerebellar fibres
  • Either at internal capsule or anterior pons

Dysarthria + clumsy hand

  • Internal capsule or anterior pons

Jauch EC, Saver JL, Adams HP Jr, Bruno A, Connors JJ, Demaerschalk BM, Khatri P, McMullan PW Jr, Qureshi AI, Rosenfield K, Scott PA, Summers DR, Wang DZ, Wintermark M, Yonas H; on behalf of the American Heart Association Stroke Council, Council on Cardiovascular Nursing, Council on Peripheral Vascular Disease, and Council on Clinical Cardiology. Guidelines for the early management of patients with acute ischemic stroke: a guideline for healthcare professionals from the American Heart Association/American Stroke Association. Stroke. 2013;44:870–947.
Lyden P, Brott T, Tilley B, et al. Improved reliability of the NIH Stroke Scale using video training. NINDS TPA Stroke Study Group. Stroke. 1994;25(11):2220-2226. doi:10.1161/01.str.25.11.2220
Lange Clinical neurology and neuroanatomy; A localization approach. Berkowitz, A. McGraw-Hill Education / Medical; 1st Edition (December 23, 2016)

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